María Generosa
Crespo Leiro
Profesora Titular de Universidad
Nieves
Doménech García
Publicaciones en las que colabora con Nieves Doménech García (18)
2024
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Reduced mitochondrial pyruvate carrier expression in hearts with heart failure and reduced ejection fraction patients: ischemic vs. non-ischemic origin
Frontiers in Cardiovascular Medicine, Vol. 11
2023
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SERCA2a Protein Levels Are Unaltered in Human Heart Failure
Circulation, Vol. 148, Núm. 7, pp. 613-616
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Soluble HLA-G levels in heart transplant recipients: Dynamics and correlation with clinical outcomes
Transplant Immunology, Vol. 76
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Survival impact of previous statin therapy in patients hospitalized with COVID-19
Medicina Clinica, Vol. 160, Núm. 1, pp. 1-9
2022
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Cardiac protection induced by urocortin-2 enables the regulation of apoptosis and fibrosis after ischemia and reperfusion involving miR-29a modulation
Molecular Therapy - Nucleic Acids, Vol. 27, pp. 838-853
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Specialized Proresolving Mediators Protect Against Experimental Autoimmune Myocarditis by Modulating Ca2+ Handling and NRF2 Activation
JACC: Basic to Translational Science, Vol. 7, Núm. 6, pp. 544-560
2021
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Extracellular Matrix in Heart Failure: Role of ADAMTS5 in Proteoglycan Remodeling
Circulation, Vol. 144, Núm. 25, pp. 2021-2034
2020
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Mitochondrial pyruvate carrier abundance mediates pathological cardiac hypertrophy
Nature Metabolism, Vol. 2, Núm. 11, pp. 1223-1231
2019
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Circulating Galectin-3 Following Heart Transplant: Long-term Dynamics and Prognostic Value
Revista Espanola de Cardiologia, Vol. 72, Núm. 11, pp. 899-906
2018
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Comparison of the PI3K-AKT-mTOR Pathway Expression Profile in Tumoral Versus Nontumoral Tissue Samples From Heart Transplant Recipients
Revista Espanola de Cardiologia, Vol. 71, Núm. 8, pp. 674-676
2017
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Role of NOD1 in Heart Failure Progression via Regulation of Ca2+ Handling
Journal of the American College of Cardiology, Vol. 69, Núm. 4, pp. 423-433
2016
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Analysis of mitochondrial proteins in the surviving myocardium after ischemia identifies mitochondrial pyruvate carrier expression as possible mediator of tissue viability
Molecular and Cellular Proteomics, Vol. 15, Núm. 1, pp. 246-255
2013
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Mitochondrial DNA haplogroup H as a risk factor for idiopathic dilated cardiomyopathy in Spanish population
Mitochondrion, Vol. 13, Núm. 4, pp. 263-268
2012
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Indoleamine, 2-3 dioxygenase activity could be an early marker of graft rejection in heart transplantation
Transplantation Proceedings
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Mitochondrial Haplogroups H and J: Risk and Protective Factors for Ischemic Cardiomyopathy
PLoS ONE, Vol. 7, Núm. 8
2005
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Humoral heart rejection (severe allograft dysfunction with no signs of cellular rejection or ischemia): Incidence, management, and the value of C4d for diagnosis
American Journal of Transplantation, Vol. 5, Núm. 10, pp. 2560-2564